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CGJ12"]
Een Jazz van mij lijdt ook aan het "Blechtrummel-syndroom" en heeft er bij 155 ook de brui aan gegeven.
Die is weliswaar nu toch stiekem doorgegroeid naar 160 (geb 27-6-2001), maar toch nog ver onder het streven..............wellicht dat ik haar nog kan aanbieden bij het NMRPS...
[/quote]

Die is dan toch pas 2??? Zie je eigen lijstje??? Wordt-ie ong. 1.65 en kun je um aan mij verkopen!!

[quote="CGJ12 schreef:

Maar 145 als 2,5 jarige?
Met een beetje mazzel blijft het zelfs een D-pony. Kan ie later FEI-proeven lopen...........

CGJ12 schreef:

Heb ik eindelijk een 80 punten merrie, kan ik wéér niet naar de NMK!

Missouri schreef:

Ach jawel! Er is nu toch ook een 4jarigen rubriek???

En anders kun je naar het NRPS

En anders heb ik hem gewoon goedkoper

_Rick schreef:

Citaat:

want een snelle groei kan schade veroorzaken zoals OCDtjes

OCDtjes ontstaan voor het jaar hoor, dus daar hoeft ze niet bang voor te zijn.

CGJ12 schreef:

standaard lijstje:
1-jaar: 150
2-jaar: 160
wordt 168/170

_Rick schreef:

Citaat:

want een snelle groei kan schade veroorzaken zoals OCDtjes

OCDtjes ontstaan voor het jaar hoor, dus daar hoeft ze niet bang voor te zijn.

Anna42"]
[quote="_Rick schreef:

Citaat:

want een snelle groei kan schade veroorzaken zoals OCDtjes

OCDtjes ontstaan voor het jaar hoor, dus daar hoeft ze niet bang voor te zijn.

Pipo"]
Volgens mij heeft de waiboerhoeve dmv een onderzoek inmiddels bewezen dat te hard groeien de kans op ocd vergroot.

Het is niet voor niets dat je OCD veelvuldig bij flink uit de kluiten gewassen paarden ziet.
[/quote]


Wobbler syndrome schijnt ook wel eens te ontstaan door te snel groeien. En wobblersyndrome wordt wel eens veroorzaakt door ODC'tjes. Deze dingen bij elkaar opgeteld klopt het wel.

[quote="Anna42 schreef:

[ OCD ontstaat tijdens de groei op jonge leeftijd en onstaat tijdens het werk.
[/quote]

Desiree"]
[quote="Pipo"]Volgens mij heeft de waiboerhoeve dmv een onderzoek inmiddels bewezen dat te hard groeien de kans op ocd vergroot.

Het is niet voor niets dat je OCD veelvuldig bij flink uit de kluiten gewassen paarden ziet.
[/quote]


Wobbler syndrome schijnt ook wel eens te ontstaan door te snel groeien. En wobblersyndrome wordt wel eens veroorzaakt door ODC'tjes. Deze dingen bij elkaar opgeteld klopt het wel.

[quote="Anna42 schreef:

[ OCD ontstaat tijdens de groei op jonge leeftijd en onstaat tijdens het werk.
[/quote]
Niet waar, tenzij je te vroeg begint met inrijden .OCD ontstaat echt in de eerste 3 levensjaren, daarna niet meer.

Citaat:

Wobbler Syndrome refers to a number of diseasestates in the horse. The most common is termedcervical vertebral malformation (CVM) and is character-ized by malformation or compression of the spinal cordwhich leads to spasticity, ataxia, and incoordination.
These symptoms are caused by damage to or compres-sion on the spinal cord.Cervical vertebral instability (CVI) and cervical staticstenosis (CSS) are the two distinct types of CVM. CVIcauses dynamic spinal cord compression.
It typicallyaffects horses from 4-12 months of age. CSS typicallyaffects horses between 12-36 months of age and ischaracterized by a closing of the cervical canal causingcompression on the spinal cord. The compression, mal-formation or lesions of CVM usually occur between thethird and fifth cervical vertebrae.
The miniature horse is the only breed that has es-caped diagnosis of the wobbler syndrome. The Thoroughbred, Quarter Horse, and Morgan are the mostcommonly affected breeds.Causes
The exact cause of the wobbler syndrome in horsesis unknown.
It is believed that the syndrome is causedby several factors: (1) genetic predisposition, (2) nutri-tional imbalances, (3) rapid growth, (4) physicaltrauma, or (5) a combination of these.
Researchers have suggested that the wobbler syn-drome is related to neck length, implying the longerthe neck, the more likely the horse is to develop thesyndrome.
Surveys have shown that males withwobbler syndrome out-number females three to one.The reason for this sex difference is not fully under-stood. However, the difference may result from the in-fluence of estrogen and testosterone on rate of growthor on the development of orthopedic disease.
The breeding of two wobbler parents does not al-ways increase the incidence of the syndrome in the off-spring. However, other metabolic bone disorders suchas osteochondrosis, physitis, and contracted tendonsare more common in the offspring of wobbler parents.
Nutritional imbalances and physical trauma are twocauses that are the easiest to control. Nutritional imbal-ances can be avoided by feeding a balanced ration thatmeets the nutrient requirements for the particular classof horse being fed. For more information on feeding,consult the nutrition series ASC 111-115.
Physicaltrauma usually results from injury, particularly when ayoung horse is tied up for the first time or when ahorse is cast in a stall.
Clinical SignsMalformation most commonly occurs between the 3-4, 4-5, and 5-6 cervical vertebrae, depending on thetype of CVM.
Lesions occur most frequently at C3-4andC4-5for CVI and compression at C5-6and C6-7for CSS.
However, malformation, lesions, and compression havebeen reported on other cervical vertebrae.These malformed or compressed vertebrae pressagainst the spinal cord and interfere with messages be-ing sent by the brain. Therefore, the interference withnormal nerve transmission from the brain to the legsleads to noticeable incoordination (ataxia). Ataxia isusually seen in the hind quarters. As the condition per-sists, weight loss and weakness may be noticeable.Clinical signs may appear suddenly or gradually, fol-lowing known or suspected trauma, and may increasein severity until death. However, death from thewobbler syndrome is unusual. The horse may show pe-riods of slight improvement, but will never completelyrecover, if aggressive treatment is not performed.
“Wobbler’s heel” is another clinical sign that occurswhen the horse is in advanced stages of the syndromeand incoordination is evident. Wobbler’s heel is charac-terized by the horse reaching forward with the backfoot and lacerating the bulbs of the heel of the frontfoot.
This could cause lameness, further complicatingthe syndrome.ASC-133Wobbler Syndrome in HorsesEquine Section, Department of Animal Sciences
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DiagnosisDiagnosis of spinal cord damage requires quality ra-diographs of the cervical region. Radiographs will re-veal signs of “lipping,” “flaring,” evidence of lesion orstenosis of the vertebrae to indicate whether the horseis suffering from CVM. A myelogram is also used toshow a static cord compression. The procedure in-volves injecting radio-opaque dye into the area sur-rounding the spinal cord in the region behind thehead. The problem with high quality radiographs and amyelogram is that they are very expensive and requirespecial equipment and expertise.Horse owners and veterinarians can perform lesscostly tests to determine whether a horse has CVM. Thefirst sign an owner may notice will be incoordinationof the rear legs. Horses can compensate with vision tocoordinate the fore limbs, but will stumble with theirhind legs because they cannot visually compensate.Turning the horse in a small circle is a quick and easytest to detect a horse with wobbler syndrome. If he issuffering from the syndrome, he will swing his rearlegs out as he makes the turn. Also, horses afflictedwith the syndrome will have trouble backing up. In-stead of letting their rear legs back up in a two beat di-agonal fashion with their front legs, they back up withtheir front legs until they get in an awkward positionand they will hop backwards with their rear legs.Two other ways of detecting wobbler syndrome areby performing the sway test and by checking theweakness of the tail. Two people are necessary to ad-minister the sway test. One person walks the horseaway while the other person grabs the tail and pullsthe horse to one side. A normal horse will allow you todo that once before rapidly correcting itself. However,a horse exhibiting wobbler syndrome cannot tell wherehis limbs are, especially his hind limbs, thus, you caneasily pull him over to one side.Another simple test used routinely is simply to pickup the horse’s tail. If there is absolutely no resistance,the horse is having some type of problem with normalnerve function in his spinal cord. Thus, the weaknessin resistance of the tail is attributed to the pressure onthe spinal cord caused by the malformation of the cer-vical vertebrae.Diagnosis of wobbler syndrome is not easy becauseseveral other causes of incoordination exist in horses,such as viruses, protozoa in the spinal cord, parasites,tumors, or fractures.TreatmentsHorses with this syndrome can be treated in differ-ent ways, either through drug therapy, surgery, or ag-gressive management. Drug therapy and surgery arevery costly and are not always practical for mosthorseowners.Certain drugs decrease the nerve tissue swelling andintracranial pressure. Some examples of these drugs in-clude osmotic agents, such as mannitol (0.5-1.0 g/kg,i.v.), dimethylsulfoxide (DMSO) (0.5 g/kg of a 40-50%solution, i.v. diluted with isotonic fluids and given bygravity flow), and diuretics, such as furosemide. Ste-roids are frequently used clinically for spinal cord andhead trauma, but experiments have shown no benefit.Those horses that cannot be helped with drugtherapy have the surgical option. Veterinary surgeonsin the United States have recently devised a surgicalprocedure adapted from human surgery called thecloward method for fusing vertebrae. The surgical tech-nique involves drilling a hole between the affected ver-tebral bodies from underneath the neck and inserting astainless steel prosthesis called a “Bagby Basket,”which fuses and immobilizes the vertebrae. The sur-gery requires specialized instruments and takes ap-proximately 1 1/2 hours to perform.In the long term, complete recovery and return tonormal athletic pursuits have occurred in 50-55% of thecases. Because of the later age of onset (2-4 years),some animals may not necessarily return to racing, butnevertheless, compete adequately in other fields.Aggressive nutritional management and controlledexercise, coupled with early diagnosis, have recentlyproven to produce excellent results in the treatment of“wobblers.” Researchers at the University of Pennsylva-nia have been very successful in treating wobbler syn-drome by substantially reducing nutrient intake andlimiting exercise. In some cases, total confinement isrequired until signs of incoordination disappear and ra-diographs show no evidence of malformations. Suchtreatment requires time and patience, but horses re-ceiving this treatment attain normal heights andweights and resume normal activity.Educational programs of the Kentucky Cooperative Extension Service serve all people regardless of race, color, age, sex, religion, handicap, or national origin.Issued in furtherance of Cooperative Extension work, Acts of May 8 and June 30, 1914, in cooperation with the U.S. Department of Agriculture, C. Oran Little,Director of Cooperative Extension Service, University of Kentucky College of Agriculture, Lexington, and Kentucky State University, Frankfort.Issued 4-92—2M